Context

Iron is vital for brain growth during fetal life and early childhood, as it supports neuronal and glial energy metabolism, neurotransmitter synthesis and myelination.1 The association of fetal-neonatal latent iron deficiency (LID) with abnormal neurocognition during early infancy has been evaluated by hippocampal event-related potentials (ERPs) for memory recognition and by auditory brainstem response (ABR) for auditory neuronal maturation.2 ,3 LID during infancy has also been associated with poor neurocognitive outcomes with impaired mental and psychomotor functions at school age.4 …